A Breakthrough in Understanding Long COVID
- People with long COVID experience persistent fatigue, breathlessness, and brain fog. Research links these effects to fibrin microclots intertwined with neutrophil extracellular traps that obstruct microvessels and impair oxygen delivery
- SARS-CoV-2 pushes blood into a hypercoagulable state, damaging endothelium, activating platelets, and inflammatory cytokines. This causes clot risk to persist for months, even after mild infections and hospital discharge
- Long COVID essentially boils down to mitochondrial dysfunction. Management includes proteolytic enzymes to help clear spike protein, while the I-RECOVER protocol guides detoxification, inflammation control, and mitochondrial repair to restore overall function
- Protecting cardiolipin, the mitochondrial membrane fat, requires limiting easily oxidized linoleic acid (LA) from seed oils, processed foods, nuts, seeds, and grain-fed meats
- Methylene blue may enhance mitochondrial respiration at low doses. Additionally, correcting copper deficiency and lowering excess iron through blood donation support energy production
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